Skip to main content

Fun Time

Comments

Post a Comment

Popular posts from this blog

REVERSE TRIGGERING: A newly classified though common form of double triggering

Reverse triggering is a recently defined type of double triggering where a controlled mechanical breath, stimulates receptors in the lung, eliciting inspiratory effort. Seen in patients who are deeply sedated with high mechanical ventilation rates. Reverse triggering could be explained by respiratory entrainment, a form of patient ventilator interaction, where diaphragmatic muscle contraction is triggered by ventilator insufflations, leading to breath initiation. Consequences of reverse triggering are large. Continuously induced muscle contraction of diaphragm cause cytokine release and muscle fibre damage. Additionally it increases inspiratory muscle load and oxygen consumption and may lead to cardiovascular instability. Reverse triggering also makes measurement of plateu pressure misleading as well as may generate high platue pressure and ventilator induced lung injury. Management is not entirely clear. Increasing sedation does not help. Increasing inspiratory time will cause the...
Post a Comment
Read more

Vocal Cord Dysfunction (VCD ) or PARADOXICAL VOCAL CORD MOVEMENT (PVCM)

▪Inappropriate adduction of true vocal cords, mostly during inspiration. This results in dyspnoea and strider during inspiration. Rarely it may happen during expiration also. ▪15 Y F presenting with acute respiratory distress for 48 hours. For the past 2 years she was on inhaled bronchodialtors and steroids with short cources of oral/IV steroids for bronchial asthma. There was history of 4 hospital admissions and several emergency visits for symptoms attributed to asthma. Examination revealed apprehensive, tachycardic, tachpnoic girl with accessory muscle use and widespread rhonchi bilaterally, SPO2 93% on room air. Other systemic examination were normal. She was started on inhalation therapy but her conditioned worsened. Oxygen saturation felled to 78% ON 10 L face mask,  ABG revealed pH 7.53, PaO2 58, PaCO2 28. She was intubated emergency and shifted TO ICU. She was treated as life threatening attack of bronchial asthma. She improved dramatically and successfully e...
Post a Comment
Read more

HYPONATREMIA: MANAGEMENT PEARLS

"The difficulty lies, not in new ideas  but escaping old ones,  which ramifies,  for those brought up with them,  as most of us has been,  into every corner of our minds" - John Maynard Keynes  Hyponatremia is defined as serum sodium concentration less than 135 mmol/L. Biochemical severity of hyponatremia has been described as mild (S. Na of 130-135 mmol/L), moderate (125- 129 mmol/L) and Severe/ profound (125 mmol/L). Clinical severity of hyponatremia is recognized based on urgency of treatment. Severe symptoms are the result of cerebral edema and increased intracranial pressure with risk herniation. It manifests as vomiting, seizures, obtundation and cardiorespiratory distress.                                Moderately severe symptoms of hyponatremia are due to mild cerebral edema but no ris...
Post a Comment
Read more